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Proposed mechanisms by which extended exposure of neuronal cultures to paclitaxel leads to desensitization of peptide release include disruption of axonal transport and subsequent impairment of the movement of organelles and/or calcium dysregulation (Boehmerle et al., 2007
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Suppression of TNF-α induced NFκB activity by gallic acid and its semi-synthetic esters: Possible role in cancer chemoprevention
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