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Indeed, neuroinflammation plays a critical role in the pathogenesis and progression of AD [69,91], and, in this section, the author has pointed to how the targeting of neuroinflammation with STS may provide an effective treatment strategy for the development and progression of LOAD by providing an anti-inflammatory approach and therapy before there is significant neuronal loss
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This study provides an early report of the loss of glial cells within the PFC of patients with mood disorders, consistent with the hypothesized glutamate abnormalities in these disorders
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