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Dietary copper restriction alone is not evidence-based or effective in these patients [71] , as intestinal copper absorption is genetically controlled through specific enterocyte transporters [72] and tightly regulated to meet the body's needs [27,72-76]
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A los 20 años, los niveles plasmáticos de GHK-Cu promedian aproximadamente 200 ng/mL, descendiendo a aproximadamente 80 ng/mL a los 60 años
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