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Clinicians will typically try standard prenatal vitamins or oral supplementation first, resorting to IM/IV B-complex therapy if those are inadequate or not feasible
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In the neonatal hypoxic–ischemic encephalopathy (HIE) model, hypoxic stress disrupts the COMMD1/ATP7A signaling axis, leading to the abnormal accumulation of Cu2+ and copper-induced cell death
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