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Targeting IGF1/IGF1r signaling relieve pain and autophagic dysfunction in NTG-induced chronic migraine model of mice | The Journal of Headache and Pain | Springer Nature Link
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These mechanisms act under both redox-sensitive and -insensitive conditions, influencing NRF2 at the transcriptional, post-transcriptional, and post-translational levels, and involving a range of proteins and epigenetic factors such as DNA methylation, histone modifications, and microRNA (Bryan et al
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