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Loss of CD98HC phosphorylation by ATM impairs antiporter trafficking and drives glutamate toxicity in Ataxia telangiectasia | Nature Communications
Endothelial dysfunction in hypertension: Current concepts and clinical implications
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33 , 35 In terms of pathology, the abnormal aggregation of α-synuclein is one of the key pathological features of PD, and more and more evidence suggests that the aggregation of this protein may originate in the gut and spread to the brain through the gut-brain axis, leading to neurodegeneration
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