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Experimental and clinical findings in the last few years suggest that ferroptosis plays a role in both chronic and acute CNS diseases via disease-specific molecular mechanisms involving iron dysregulation, antioxidant deficiency, mitochondrial dysfunction and neuroinflammation
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Palmitoyl-CoA levels were decreased, but no clear tendency was observed for expression of different members of the acyl-CoA-synthetase family (Figure 5 and Supplemental Figure 4B)
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