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Through its angiogenic properties, BPC-157 promotes the growth of new blood vessels, significantly enhancing the healing of the myotendinous junction, which is typically a weak spot due to its poor vascularization [24, 38]
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Benoist, Kawas LH, and Harding, JW, unpublished data) have recently demonstrated that both Nle 1 -AngIV and dihexa bind HGF, leading to its activation, and that the procognitive and/or synaptogenic actions of these compounds are blocked by both HGF and c-Met antagonists
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