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NMDARs are remarkable because they serve a dual function as regulators of both synaptic communication and long-term changes in synaptic strength, such as long-term potentiation and depression (long-term potentiation and long-term depression) of AMPAR-mediated synaptic signaling (Rebola et al., 2010
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In five separate experiments using hepatocytes prepared from different human livers, nitrobenzene at concentrations up to 1 mmol/litre was negative for induction of unscheduled DNA repair (Butterworth et al., 1989)
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