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These findings suggest that G93A-SOD1 epSPCs undergo a metabolic rewiring characterized by reduced glucose oxidation and a compensatory reliance on glutamine anaplerosis to sustain TCA cycle activity and antioxidant defense

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37 , 38 Different metals may assume complementary catalytic functions, one metal center may facilitate H 2 O 2 decomposition or Fenton like hydroxyl radical generation, whereas another may promote oxygen activation, GSH depletion, copper ion cycling, or mitochondrial metal stress